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Analyses were adjusted for age, sex, T2D duration, semaglutide dose, baseline endpoint level, index year (to account for reimbursement policy changes), and concomitant anti-diabetic treatment
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As there is no meaningful expression of GLP-1 receptors on hepatocytes, the major effect of GLP-1 agonists on MASH is likely to be indirect and may be related to a reduction in calorie intake, body weight and insulin resistance, all of which would lead to reduced liver lipid accumulation and hepatic inflammation 7,8
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